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School of Biomedical Sciences School of Biomedical Sciences
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Dr Seung Jae Kim

Postdoctoral Research Fellow
School of Biomedical Sciences
seungjae.kim@uq.edu.au
View researcher profile

Publications

Journal Articles (6)

Journal Articles

Dodd, Garron T., Kim, Seung Jae, Méquinion, Mathieu, Xirouchaki, Chrysovalantou E., Brüning, Jens C., Andrews, Zane B. and Tiganis, Tony (2021). Insulin signaling in AgRP neurons regulates meal size to limit glucose excursions and insulin resistance. Science Advances, 7 (9) eabf4100. doi: 10.1126/sciadv.abf4100
Kakall, Zohra M., Cohen, E. Myfanwy, Farnham, Melissa M. J., Kim, Seung Jae, Nedoboy, Polina E. and Pilowsky, Paul M. (2019). Integration of hindbrain and carotid body mechanisms that control the autonomic response to cardiorespiratory and glucoprivic insults. Respiratory Physiology and Neurobiology, 265, 83-91. doi: 10.1016/j.resp.2018.08.008
Cohen, E. Myfanwy, Farnham, Melissa M.J., Kakall, Zohra, Kim, Seung Jae, Nedoboy, Polina E. and Pilowsky, Paul M. (2018). Glia and central cardiorespiratory pathology. Autonomic Neuroscience: Basic and Clinical, 214, 24-34. doi: 10.1016/j.autneu.2018.08.003
Kim, Seung Jae, Fong, Angelina Y., Pilowsky, Paul M. and Abbott, Stephen B. G. (2018). Sympathoexcitation following intermittent hypoxia in rat is mediated by circulating angiotensin II acting at the carotid body and subfornical organ. Journal of Physiology, 596 (15), 3217-3232. doi: 10.1113/JP275804
Kim, Seung Jae, Pilowsky, Paul M. and Farnham, Melissa M. J. (2016). Intrathecal intermittent Orexin-A causes sympathetic long-term facilitation and sensitizes the peripheral chemoreceptor response to hypoxia in rats. Journal of Pharmacology and Experimental Therapeutics, 358 (3), 492-501. doi: 10.1124/jpet.116.234443
Kim, Seung Jae, Kim, Yeon Jae, Kakall, Zohra, Farnham, Melissa M. J. and Pilowsky, Paul M. (2016). Intermittent hypoxia-induced cardiorespiratory long-term facilitation: a new role for microglia. Respiratory Physiology and Neurobiology, 226, 30-38. doi: 10.1016/j.resp.2016.03.012
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